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In conclusion, although there is substantial evidence in animals that numerous selenoproteins (e.g., mGPX4, nGPX4, GPX5, TXNRD3, selenoprotein P) are crucial for normal male reproductive function, and that their deficit (genetic knockdown) leads to reduced or total infertility, the studies in humans are still scarce

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Beneficial effects of -sitosterol on type 1 cholecystokinin receptor dysfunction induced by elevated membrane cholesterol