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The major pathophysiological component of cerebral ischemic-hypoxic injury is the production of large amounts of free radicals, which further damage lipids, proteins, and deoxyribonucleic acid (DNA), thereby inducing neuronal cell death[48]

9 (29), eadg2697 (2023)

The experiments were conducted in accordance with institutional protocols and adhered to the Guide for the Care and Use of Laboratory Animals (NIH Publication No

Worldwide Racial and Ethnic Distribution of Alpha1-Antitrypsin Deficiency: Summary of an Analysis of Published Genetic Epidemiologic Surveys. Chest, vol