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Based on these new insights, a theory has been proposed that explains the pathogenesis of MASLD through processes that exceed the normal metabolic capacity of the liver, leading to the excessive accumulation of toxic lipids, hepatocellular stress, injury, and cell death [40]

For instance, quercetin inhibits NETs formation by targeting the P2X7R/P38MAPK/NOX2 signaling pathway (105), thereby alleviating oxidative stress and inflammatory responses in neutrophils

E3 ubiquitin ligases often act with remarkable specificity, targeting substrates in a time- and compartment-dependent manner

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