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These findings suggested that VNS may ameliorate hepatic I/R injury-induced AKI by suppressing inflammation, oxidative stress, and apoptosis probably through activating the Nrf2/HO-1 signaling pathway

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The lack of oxygen due to cessation of blood flow results in depleted ATP stores, thereby disrupting the ionic gradients responsible for regulating neuronal firing, resulting in increased action potentials and aberrant glutamatergic signaling