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Indeed, disassembly or conformational rearrangement of a proportion of functional molecules on infectious virions, by exposing epitopes that are not available on functional molecules, presents opportunities for nnAbs to trigger effector systems such as complement activation and phagocytosis

The authors concluded that the senescent microenvironment maintained by SASP and p53-pS15 signalling promotes keloid aggressiveness and relapse, and that FOXO4-DRI targets this mechanism selectively

The evidence did not establish effectiveness, and recognised evidence-based withdrawal treatments are available