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This phenotype is reminiscent of that observed for pU S 3, which modulates NEC function during viral budding but leads to an accumulation of perinuclear virions in herniations when defective (see introduction)

Patra JK, Das G, Fraceto LF, Campos EVR, Rodriguez-Torres MDP, Acosta-Torres LS, et al

* Correspondence: Haixia Zhou, [email protected] These authors have contributed equally to this work Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

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