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Conversely, clinical studies have demonstrated that GLS1 promotes mitochondrial respiration rate and tumour growth potential [17]

In 2022, scientists proposed the concept of cuproptosis, which is a regulatory mode of death induced by intracellular copper loading exceeding the threshold maintained by homeostatic mechanisms [183]

11,12 However, the inhibition of the xCt cystine/glutamate antiporter failed to trigger ferroptosis, despite its recognized role as a key regulator of this process