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This was attributed to the higher intracellular glutathione content in the #14 mutant when cultured in the molasses/CSL-based medium (2.6%), 87% higher than in the YP-based medium

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This effect seems to be independent of mu opioid peptide receptors (MOP), as it was not reversed by the MOP antagonist naltrexone, nor mimicked by the MOP agonist methadone [65]

Chelation of copper or genetic deletion of this copper-binding site inhibits autophagy and hence reduces the fitness of KRAS-induced cancers