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The divalent iron (Fe 2+ ) generated through intestinal absorption or degradation by red blood cells is oxidized to trivalent iron (Fe 3+ ), which becomes circulating iron by binding transferrin (TF) to form TF-Fe 3+ (Frazer and Anderson, 2014)

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288,289 In diabetic conditions, these macrophages can transition to a proinflammatory phenotype, accumulate, and contribute to nerve damage