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Maintenance of these structures are crucial, because the dysregulation of tight junction proteins during injurious events, such as in celiac disease or intestinal ischemia, and the inability to repair tight junction structures after injury can result in intestinal inflammation, sepsis, and multiple organ dysfunction in clinical patients (16, 17)

GPS, with its anti-inflammatory and antioxidant properties, contributes to creating an optimal microenvironment for wound recovery

Furthermore, the NPs can be readily surface-modified with diverse organic compounds, ligands, and other agents to achieve target specificity, augmenting their efficacy

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