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At present, the exact mechanism by which heavy metal exposure/environmental factors induce epigenetic modifications is not known

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lipopolysaccharide) HO-1 is recruited to the cell membrane caveolae through p38 MAPK-dependent mechanism, blocking the activated proinflammatory signaling cascade (142)

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Diseases secondary to defects in intergenomic communication These diseases are due to defects in nuclear factors involved in the replication, maintenance, and translation of mtDNA

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The publication of this article was funded by the Qatar National Library

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