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[Abstract]2022 Aug;54:102392

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AX has been shown to buffer mitochondrial overload, which can be induced by physical exercise, as exercise inherently promotes ROS formation and mobilizes fuels within the mitochondria, but also by an HFD, as observed in obesity, which continuously stimulates the mitochondrial network in ATP production, fatty acid synthesis, and the consequent generation of ROS (Figure 2) [4,46]