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Suppression of HMOX1 can effectively rescue the negative effects of high glucose and high lipids by rebalancing iron and oxidative stress and blocking excessive ferroptosis (66)

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This enzyme lies at the bottom of the transulfuration pathway and requires FAD cofactor for the utilized, oxidized form of glutathione (GSSH) to re-constitute itself back to its reduced active (GSH) form in order to be useful in antioxidant defense (Figure 1) (62)