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From our experience, these relate mainly to the following difficulties: (1) Clinical documents are variously structured, notational, lengthy, and incomplete

This is clearly demonstrated by experiments in which the administration of the neurotoxin MPTP, which blocks Complex I of the mitochondrial electron transport chain, induces characteristic features of PD in mice [56, 57]

Rather than activating the growth hormone receptor, AOD 9604 functions as a pathway-specific modulator of lipid metabolism, enabling investigation of lipolysis and energy regulation in experimental systems without engagement of IGF-1mediated endocrine signaling

In this context, artificial intelligence (AI) and machine learning (ML) have emerged as transformative tools for modeling ROS biology and therapeutic design [311]