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These findings together imply that upregulation of Prxs renders motor neurons less susceptible to neurodegeneration, whereas breakdown of this redox system at late disease stages induces neuronal degeneration and accelerates disease progression (Kato et al., 2005)

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bile acids in portal circulation are further taken up by the liver through sodium-dependent taurocholate co-transporting polypeptide (NTCP, also known as SLC10A1, and this protein can act as a receptor for HBV) and sodium-independent transporter protein organic anion-transporting polypeptide C (OATP-C, also known as SLC21A6, LST-1, or OATP2) for re-secretion into bile

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