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However, some cutting-edge technologies like PROTACs (proteolysis-targeting chimeras), RNA interference, clustered regularly interspaced short palindromic repeats (CRISPR)/Cas9-based genome editing, and artificial intelligence (AI)-based drug design, allow screening of therapeutics targeting almost all kinds of molecules, like proteins and epigenetic regulators

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267 Both mTORC1 and mTORC2 contribute to GBM progression, with mTORC1 promoting glycolysis via HIF-1 activation and mTORC2 enhancing tumor cell motility through RICTOR overexpression