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Inhalation of aerosols or mists containing the bacterium is presumed to be the primary means of acquiring legionellosis

Research Findings NAD has been studied across mitochondrial/cellular, molecular, and systemic preclinical models, with peer-reviewed laboratory literature characterizing its role as a redox cofactor and signaling substrate for sirtuin deacylases, PARP enzymes, and CD38 ectoenzymes in in-vitro and animal model settings