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Finally, despite recent advances in understanding the interaction mechanisms between EC and other polyphenols with gut microbiota, key functional strains and core metabolic enzymes remain incompletely characterized

The absence of the GST monomer with excess GSH further suggests that the dimer would be the predominant protein present

It was shown that aged or/and damaged RBCs are sequestrated in the hepatic sinusoid in a PS-dependent way, and the human sinonasal epithelial cells (HSECs) mediate the recognition and the binding of damaged RBCs in a PS-dependent manner via stabilin-1 and stabilin-2 (pro-phagocytic signals and ligandligand interactions) [275,276,277]

Vondenhoff et al., 2011) (see Figure 4A for a schematic overview)