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Oxidative stress, a crossroad between rare diseases and neurodegeneration

This regulation alleviates oxidative damage, inflammatory responses, and mitochondrial dysfunction, thereby reducing neuronal death and enhancing the therapeutic efficacy in stroke (170)

On the one hand, activation of HIF-1 in osteoblasts has been shown to suppress osteoclastogenesis by upregulating osteoprotegerin (OPG) and interleukin-33 (IL-33), thereby inhibiting the RANKL/Notch1 signaling pathway (Kang et al., 2017)