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2011 May 2;7890

Although this specific mechanism remains to be directly validated in diabetic kidney disease, it is reasonable to hypothesise that aloe-emodin may exert similar effects given the shared pathological features between diabetic kidney disease and radiation-induced tissue injuryincluding enhanced glycolytic flux, lactic acid accumulation, endoplasmic reticulum stress, mitochondrial dysfunction, and activation of the fibrosis transcriptional programme [14, 16, 17]

The proposed mechanisms are as follows: rebuilding the level of 5-HT