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Concurrently, ACC inhibition reduces inflammatory macrophage recruitment through blockade of chemokine receptors CCR2/CCR5 (e.g., via cenicriviroc), thereby attenuating pro-inflammatory cytokine production in pulmonary niches (Tacke and Weiskirchen, 2018)

Neuropharmacology 2007

These findings suggest that EA treatment mitigates the worsening of symptoms following TBI, potentially by inhibiting TGIF-mediated apoptosis in secondary brain injury post-TBI

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