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Given the prominent role of inflammation, oxidative stress, and mitochondrial dysfunction in MS pathophysiology, metabolic interventions aimed at enhancing mitochondrial function and providing an alternative energy source, such as the KD, have been explored as potential adjunctive strategies to slow disease progression and improve clinical outcomes (Table 5)

Wu et al., 2022), while some other studies showed that there were no associations (Stycze et al., 2016

This leads to enhanced production of NO, a potent vasodilator that improves endothelial function and vascular tone

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