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Although GSH may function as a dynamic determinant of intracellular copper bioavailability rather than merely a passive buffering pool (Fig

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However, TGF-1 has two sides: In addition to being an inducer driving fibrotic and anti-inflammatory responses, its inactivation may also mean exacerbating proinflammatory effects, suggesting that antifibrotic intervention that directly targets TGF- may trigger other unknown risks (145)

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STC2 activates PRMT5 to induce radioresistance through DNA damage repair and ferroptosis pathways in esophageal squamous cell carcinoma

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