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Hispidulin has been indicated with diverse mechanistic insights in cell cycle arrest: in gastric cancer AGS cells, it sustains NAG-1 (NSAID-activated gene-1) expression through ERK activation, followed by the downregulation of cyclin D1/E, and eventually induces G1/S arrest with apoptosis (Yu et al., 2013

Thus, analogous with the therapy effects noted in the counteraction of the severe occlusion/occlusion-like syndromes [18,19,20,21,22,23,24,25,26,27,28,29,30], such consistent results with a correspondingly wide range of the agents efficacy (gng) support each other for further therapy use [11]

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Given the gut microbiota-dependent production of PAGln, we next performed targeted metabolite profiling on plasma and other cardiovascular-related organs