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Phenylalanine (Phe) is an essential AA in mammals, and its conversion into Tyr is crucial for the production of thyroid hormones and catecholamines

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The surplus of ROS represents a trigger for programmed cell death processes and promotes the release of pro-inflammatory cytokines, which are also responsible for further increasing ROS levels, thus perpetuating the pathophysiological process of AD

Conclusion: ScRNA-seq and single cell IMC determined that steatotic livers have immunosuppressive TIME compared to non-steatotic livers, which is regulated by M2TAMs, T cells, CAFs, and CRC cells with the expression of PD-L1, PD1, CD44, and YAP