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pylori G27, MOI 50, 10 h) and uninfected AGS cells, normalized by the total number of live AGS cells in each condition (right)

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These results show that the IF-mediated increase in the inflammatory phenotype of eWAT was mainly determined by an increase in LAM abundance, which was largely dependent on intact p53 signalling in adipocytes of intermittently fasted HFD-fed mice

Past studies have shown that various pathways associated with the initiation of ferroptosis ultimately converge to suppress cystine import through the inhibition of cystine-glutamate antiporter (system Xc ), which is reportedly necessary for glutathione biosynthesis (Dixon et al