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First, while oral administration of glutathione in experimental animals seems to be useful in increasing plasma levels, such oral supplementation in humans is controversial, as -glutamyl transpeptidase, an intestinal enzyme, degrades exogenous glutathione before it could be absorbed (Allen and Bradley, 2015)

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By acting as a hepatocyte growth factor (HGF)/c-Met agonist, it promotes neuroplasticity, memory formation, and potential repair of damaged neural circuits

Overall our data indicate that glutathione metabolism is differently affected in the two resistant strains and that glutathione system may play an important role in the global organization of C.albicans cells for resistance to fluconazole