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Mechanism of Long-Term High-Dose Prednisolone Administration Producing Myocardial Fibrosis in Beagle Dogs

doi: 10.1038/sj.bjp.0704047

At the molecular level, they modulate inflammation-related gene expression by altering membrane phospholipid composition, disrupting lipid raft integrity, inhibiting nuclear factor kappa B (NF-B), and activating PPAR- , thereby promoting anti-inflammatory mediators and suppressing inflammation (18)