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Phenotypes resembling human ALS have been successfully replicated in SOD1 mutant mice and through the targeted deletion of the hypoxia response element (HRE) from the promoter of the mouse VEGF gene (Thakur et al

[1] Common research areas include: Tendon, ligament, and muscle repair models Gut lining healing and IBD research Nitric oxide and growth-factor (VEGF, EGF) signaling studies Nerve protection and brain-injury models Anti-inflammatory pathway research (NF-B) Bone healing and fracture repair studies How It Works BPC-157 works through several repair pathways at once