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SPNs were also recently investigated for the first time for detecting early synaptic transmission alterations exerted by -syn oligomers, combining multiple techniques like electrophysiology optogenetics, immunofluorescence, and molecular and behavioral analyses [106]

Deficiencies in CBS activity caused by genetic mutations of the CBS gene are the most frequent cause of familial HHCy

Copper deficiency impairs copper-dependent enzymes, whereas copper overload promotes excessive reactive oxygen species (ROS) generation, leading to oxidative damage to lipids, proteins, and DNA [5, 6]