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The therapeutic potential of ferroptosis extends across diverse malignancies, necessitating future investigations into targeted delivery systems for ferroptosis inducers, synergistic treatment modalities, and dosage optimization to enhance therapeutic outcomes while minimizing adverse effects, thereby facilitating the clinical translation of ferroptosis-based therapies

Keywords : Set7/9, ROS metabolism, oxidative stress, p53, chromatin, diabetes, myocardial ischemic injury, nephropathy Reactive oxygen species (ROS) include the superoxide anion (O 2- ), hydrogen peroxide (H 2 O 2 ) and the hydroxyl radical (HO), which are produced during the normal life cycle of cells [1]

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