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In sepsis-associated cardiorenal syndrome type 5, exogenous NaHS and S-propargyl-cysteine (SPRC) activate this pathway to clear damaged mitochondria, restore membrane potential, suppress ROS generation, and promote RAW264.7 macrophage M1-to-M2 transition in cardiac/renal tissues ( 2 S donors and endogenous stress responses to macrophage phenotypic homeostasis

For DB, NGM plates were supplemented with bactericidal antibiotic mix (50 g ml 1 kanamycin and 100 g ml 1 carbenicillin)

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