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30 min pretreatment, followed by 24 h AO exposure) dose-dependently inhibits the release of proinflammatory mediators (TNF-, IL-1, NO) induced by AO in primary mouse microglia, suppresses oxidative stress, and restores the antioxidant capacity of cells [5]

It modulates gut microbiota and faecal metabolites, cutting harmful fatty acids

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The copper ion sits in a square-planar pyramid configuration, creating an extraordinarily high binding affinity approximately 10 million times higher than other tripeptides