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The intrinsic ability of cancer cells to tolerate and recover from this DNA damage can stem from several pathways, including anti-apoptotic mechanisms, cell cycle arrest, DNA damage repair (DDR) and nucleotide metabolism, the oxidative/electrophilic stress response to free radicals, autophagy

DHEA levels decline with age

The above findings suggest that ferroptosis plays a vital role in the pathology of CD through mitochondrial function deteriorates and contributes to the development of Cardiac Diseases

Significantly, it has advanced into clinical trials for conditions including mitochondrial myopathy, autosomal genetic diseases, mitochondrial genetic diseases, heart failure, myocardial reperfusion injury, renal reperfusion injury, and age-related skeletal muscle issues