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NAD is a critical cofactor for OXPHOS and sirtuin-mediated deacetylation processes, which are essential regulators of mitophagy [22]

Although 4 frequency does not consistently differ between patients and controls, it has been linked to symptom severity, poorer outcomes, suggesting a role in shaping positive symptom expression

Discussion In the present investigation, we demonstrate for the first time that the decreased glutathione-mediated redox/antioxidant capacity previously observed in plasma and immune cells from children with autism is also significantly decreased in two brain regions previously shown to be affected in autism, the cerebellum and BA22

The hepatic parenchymal cells, the hepatocytes, have an immensely broad range of synthetic and catabolic functions, which are summarized in Table 30.1