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(2017) using a human microphysiological system (i.e., organs-on-a-chip) model, demonstrated that TMAO could penetrate the blood-brain barrier

MTT 1-(4,5-Dimethylthiazol-2-yl) 3,5-diphenylformazan reagent HGF-dependent proliferation was determined by subtracting the basal proliferation (in the absence of HGF) from total proliferation rates in groups containing HGF

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