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Under inflammatory conditions, pro-inflammatory cytokines [e.g., interferon-gamma (IFN-)] can activate IDO1, diverting Trp metabolism toward the kynurenine (KYN) pathway while inhibiting 5-HT synthesis, resulting in serotonin depletion and exacerbation of neurotransmitter imbalance (Rus et al., 2015)

Clinical efficacy, however, is only as reliable as the manufacturing process that produces the liposomal GSH API behind every finished formulation

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Disruptions in redox balance, whether toward excessive oxidation or reduction, are generally deleterious to cellular function