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Inhibiting the elevated expression of GJ components in hepsin / mice reduces APAP toxicity by limiting oxidative stress in the liver To investigate GJ expression variations after APAP administration in wild-type and hepsin / mice, immunofluorescence staining for Cx32 was assessed at different time points

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Against this background of potential systemic toxicity, monitoring indicators closely linked to the core mechanisms of ferroptosis/cuproptosis and metal ion metabolism become particularly critical

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