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We have shown that effective pharmacologic inhibition of GPX4 in PTC cells with direct inhibitor RSL3 induces a robust activation of ferroptosis, increased DNA damage, impaired DNA damage repair response due to reduced p-NPM1 levels, decreased cell viability and cell migration, as well as suppressed spheroid formation and viability in vitro

Recovery timeline During infusionPatients rest comfortably while the medical team monitors the session

These beneficial effects were observed in a dose-dependent manner, emphasizing YSTJs ability to modulate inflammatory pathways and promote nerve repair (Zhang Y