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45 Collectively, NSAIDs represent the first-line strategy for targeting the inflammatory phenotype of OA by directly suppressing cyclooxygenase-driven prostaglandin pathways, forming a mechanistic foundation for more advanced anti-inflammatory interventions

Research using GHK-Cu liposome formulations in murine scald wound models demonstrated significantly elevated VEGF expression in treated wounds compared to controls, with corresponding increases in capillary density and wound closure rates