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In this context, SHMT2 initially functions as a scaffold protein that recruits BRISC to ubiquitinated IFNAR1, promoting K63-linked deubiquitination and preventing receptor degradation to sustain type I interferon signaling [130]

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These receptors are GIP (Glucose Inhibitory Peptide or Glucose-Dependent Insulinotropic Peptide) and GLP (Glucagon-Like Peptide-1)

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