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On the pharmacology side, HGF-induced MET activation exerts beneficial neuroprotective effects also in adulthood, specifically in neurodegenerative disease, and in preclinical models of cerebral ischemia, spinal cord injuries, and neurological pathologies, such as Alzheimers disease (AD), amyotrophic lateral sclerosis (ALS), and multiple sclerosis (MS)

Still, FH variants in HLRCC patients lead to loss of function, and the consequent LOH in the tumour tissue causes the complete loss of its enzymatic activity [20]

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Smoking, a direct source of oxidative stress