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COPZ1 knockdown in glioblastoma cell lines (U87MG, U251, and P3) induces ferritinophagy, upregulates NCOA4, and activates autophagy-related protein ATG7, leading to excessive Fe 2+ release, ROS accumulation, and ferroptotic cell death [36]

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In the Second Opinion Physician model, impaired disposal of homocysteine, adenosine and related products may help maintain SAH accumulation

Targeting the Nrf2-Heme Oxygenase-1 Axis after intracerebral hemorrhage