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Recent studies suggest that APR-246 may also induce cell death by depleting intracellular GSH levels

The high E-values indicated a very low residual confounding in these models

Journal of Clinical Biochemistry and Nutrition, 48 (1), 26-32

Moreover, these ion channels are key regulators of cellular electrical excitability, and their dysfunction correlates with many neurological and cardiovascular manifestations associated with HHcy