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Description

On the other hand, HO-1 overexpression causes the accumulation of intracellular iron, promotes lipid peroxidation and triggers ferroptotic cell death (61,72,73)

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NOX-4 has been found to be involved in inflammation in diabetes and its deletion resulted in the downregulation of Nuclear factor-kappa B (NF-B) signaling, an important mediator of inflammation (Jha et al

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Glyoxalase 1 and its substrate methylglyoxal are novel regulators of seizure susceptibility

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The use of nanoparticles as delivery vehicles solves these problems by improving the efficacy of genome editing while minimizing cell damage due to their small size

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